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Faculty Detail    
Campus Address THT 1030 Zip 0000
Phone  (205) 975-7919
Other websites Google Scholar

Undergraduate  North Carolina State University    2005  BS 
Graduate  North Carolina State University    2007  MS 
Graduate  Wake Forest University    2011  PhD 

Faculty Appointment(s)
Appointment Type Department Division Rank
Primary  Surgery   Surgery - Gen Surg Trauma Section Assistant Professor
Secondary  Dept of Biomedical Engineering  Dept of Biomedical Engineering Assistant Professor

Graduate Biomedical Sciences Affiliations
Pathobiology and Molecular Medicine 

Biographical Sketch 
Dr. Jill Richter is an Assistant Professor of Surgery in the Division of Acute Care Surgery at UAB. Dr. Richter is a biomedical engineer by training, and she received her B.S. and M.S. degrees from North Carolina State University. For her doctoral work, Dr. Richter attended the School of Biomedical Engineering and Sciences, a joint program between Wake Forest University and Virginia Tech, and she completed her research training at the Wake Forest Institute for Regenerative Medicine. Dr. Richter trained as an NIH T32 Postdoctoral Fellow at the University of Cincinnati in the Department of Surgery. After moving to UAB in 2013, Dr. Richter completed additional postdoctoral fellowships in the Departments of Radiology, Pathology and Anesthesiology. Her faculty appointment at UAB began in 2016.

Society Memberships
Organization Name Position Held Org Link
Shock Society  Member   
Eastern Association for the Surgery of Trauma  Member   
Biomedical Engineering Society  Member   

Research/Clinical Interest
Endothelial Glycocalyx as a Mechanoregulator of Post-Traumatic Lung Injury
My research program is focused on understanding the role of the endothelial glycocalyx in regulating injury severity and outcomes in trauma patients. In combination with established animal models of trauma-hemorrhage and resuscitation (THR), we utilize custom-designed bioengineered cell culture systems to study the effect of hemodynamic forces on THR-related endotheliopathy, microvascular dysfunction and organ injury. Our research efforts are aimed at the identification of therapeutic strategies to prevent glycocalyx damage or restore loss of glycocalyx function caused by THR.

trauma, hemorrhage, resuscitation, vascular biology, organ failure, blood transfusion, plasma, whole blood, platelets, endothelial glycocalyx layer, lung injury, bioengineered in vitro models, mechanotransduction